How Zika Virus Evades Antiviral Response of Cells

Researchers have identified a mechanism by which Zika virus can escape detection by host cells, offering a new target for therapeutics.

Regulation of antiviral innate immune signaling and viral evasion following viral genome sensing

Zika virus antagonizes interferon response in patients and disrupts RIG-I–MAVS interaction through its CARD-TM domains, Cell & Bioscience

Type III Interferons Produced by Human Placental Trophoblasts Confer Protection against Zika Virus Infection - ScienceDirect

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TLR3 Activation by Zika Virus Stimulates Inflammatory Cytokine Production Which Dampens the Antiviral Response Induced by RIG-I-Like Receptors

5-Hydroxymethylfurfural Enhances the Antiviral Immune Response in Macrophages through the Modulation of RIG-I-Mediated Interferon Production and the JAK/STAT Signaling Pathway

IL-27 signaling activates skin cells to induce innate antiviral proteins and protects against Zika virus infection

Zika virus elicits inflammation to evade antiviral response by cleaving cGAS via NS1‐caspase‐1 axis

Transcriptional Changes during Naturally Acquired Zika Virus Infection Render Dendritic Cells Highly Conducive to Viral Replication - ScienceDirect

Biomedicines, Free Full-Text

Zika virus antagonizes interferon response in patients and disrupts RIG-I–MAVS interaction through its CARD-TM domains, Cell & Bioscience

American-Asian- and African lineages of Zika virus induce differential pro-inflammatory and Interleukin 27-dependent antiviral responses in human monocytes - ScienceDirect

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The role of NK cells in fighting the virus infection and sepsis

Cross-Reactive Dengue Virus Antibodies Augment Zika Virus Infection of Human Placental Macrophages - ScienceDirect