IJMS, Free Full-Text
Por um escritor misterioso
Last updated 07 julho 2024
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Uterine fibroids (UFs) are monoclonal, benign tumors that contain abnormal smooth muscle cells and the accumulation of extracellular matrix (ECM). Although benign, UFs are a major source of gynecologic and reproductive dysfunction, ranging from menorrhagia and pelvic pain to infertility, recurrent miscarriage, and preterm labor. Many risk factors are involved in the pathogenesis of UFs via genetic and epigenetic mechanisms. The latter involving DNA methylation and demethylation reactions provide specific DNA methylation patterns that regulate gene expression. Active DNA demethylation reactions mediated by ten-eleven translocation proteins (TETs) and elevated levels of 5-hydroxymethylcytosine have been suggested to be involved in UF formation. This review paper summarizes the main findings regarding the function of TET enzymes and their activity dysregulation that may trigger the development of UFs. Understanding the role that epigenetics plays in the pathogenesis of UFs may possibly lead to a new type of pharmacological fertility-sparing treatment method.
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non sense codon –
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IJMS Free Full-Text Research Advances In Mechanical, 52% OFF
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Ijms Free Full Text Preliminary Rna Seq Analysis Of Long Non Coding 55677
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IJMS, Free Full-Text, Autotaxin-LPA-LPP3 Axis in Energy Metabolism and Metabolic Disease, HTML
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IJMS Free Full-Text Research Advances In Mechanical, 54% OFF
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IJMS, Free Full-Text
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IJMS, Free Full-Text
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IJMS, Free Full-Text
Ijms Free Full Text Neural And Hormonal Basis Of 49680
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